Immune Hypothesis for Schizophrenia
The immune hypothesis of schizophrenia proposes that the immune system plays a role in the development of the disorder. It suggests that an overactive and/or aberrant immune response may be involved in the onset and/or progression of schizophrenia, leading to changes in the brain and the onset of psychotic symptoms. Recent research has provided support for this hypothesis, suggesting the involvement of both innate and acquired immune pathways in the development of schizophrenia. Evidence suggests that immune system abnormalities, such as inflammation, autoimmune responses and infectious agents, may interact with genetic and environmental factors to contribute to the development of schizophrenia. More research is needed to fully understand the role of the immune system in schizophrenia and to develop potential new treatments for this mental disorder.
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